Unlike conventional drug treatments for Alzheimer’s disease, coconut oil and water may help resolve some of the root causes of neurodegenerative conditions.
Have you ever noticed that sometimes a food that nourish a particular organ often look uncannily similar to it? For instance, the walnut shell and bi-hemispheric flesh look awfully like the skull and brain it is now known to be exceptionally good for. Pomegranate fruit, the actual fruiting ovary of the plant, looks a lot like the mammalian ovary that it has been proven to support, containing relatively high levels of the actual bioidentical estrogen estrone. So too is the case with the head-like coconut, which is increasingly being looked at for preventing and/or improving brain dysfunctions such as Alzheimer’s disease (AD) due to its wide range of potentially therapeutic properties, such as:
1) Increasingly energy production in the brain by providing neurons an alternative to glucose, namely, the brain-nourishing metabolites of medium chain fatty acids known as ketone bodies.
2) By contributing potent antioxidant polyphenols that may reduce oxidative stress, improve brain blood circulation and reducing neuroinflammation
3) By inhibiting amyloid beta plaque-associated neuronal toxicity.
Indeed, a growing number of cell, animal, human and population-based studies reveal that coconut consumption may provide a natural solution to a growing global epidemic that pharmaceutical drugs have been shown to do nothing to mitigate. In fact, we have discussed in previous articles how modern drug-based treatment for AD actually is more like chemical warfare than a therapeutic intervention — likely doing far more good than harm.
One reason why conventional interventions are so ineffective is that the brain is highly protected against the entry of chemicals due to the brain-blood-barrier (BBB), with most small molecule drugs and virtually all large molecule drugs incapable of gaining entry to the brain because of it. Even naturally occurring biomolecules such as fats have a hard time penetrating directly through the BBB. Coconut may be an exception to this rule, as it has copious amounts of the medium chain triglyceride known as caprylic acid, which preliminary research indicates is able to pass through the BBB and which has been demonstrated to possess anti-convulsive and neuroprotective properties in the animal model.